University:
Camosun College
Course:
NURS132 | Promoting Health and Wellbeing
Academic year:

2023
Views:

118

Pages:

3
Author:

Aysha A

LOBAR PNEUMONIA Fibrinoseppurative consolidation of large portion of a lobe or entire lobe of the lung. ✓ char. & dit fuse in Hammation att the lower lobes morth. divided into CROR MORPHOLOGICAL STAGES CONGESTION • lasts for 24 hours. • GROSS:- Involved loke of lung is heavy, boggy, and red. Blood stanel frothy fluid oozes from the cut surface MIKROSCOPY:- - Dilation & congeskom of capillaries in the Alveolar walls Air spaces M the alveoli fillel with pale eosinophila Fluid with few neutrophils, red cells, and numerous bacteria. RED HEPATI ZA TION olaste for 2-3 days Gross - lobe appears disking Red, firm, airless. Firm consistery, resembles that of Lives Pleura show SEROFIBRINOUS PLEURISY Cut section shows red and granits apperance. MIKRO SCOPY Alveoli show exudate & Interlacing strand of fibind - Numerous neutrophils & Rod cells found in filarm Neshwak GREY HEPATIZATION 5-7 days Gross:- Affected lobe loses its red color and assumes. grey appearances. Cut surface shows gray, day ond granuler app MICROS copy - Progressive disinkgration of Red calls - Fibrinomppurative exudate in the alveoli in which neutrophils Macrophages are replaced by transparent space betweer alveolor wall and exudate due to contraction of fibrin biread present in exudate RESOLUTION occurs by LIQUEFACTION of prev. solid fibrinoes constituents of exudat in air spaces o to form granuler & semifluid debris Liquefaction is due to fibrinoglic enzyme liberated from Dentrophils GRoss:- affected loke becomes more crepitant as airspace Cut sections: App trothy reopen Microscopy: Alvedor space shows granules material 2 semifluid debris COMPLICATION LONG ABSCESS- May develop with extensive tissue destructor and necrosis. 2. ORGANIZATION Pelayed, in complete resolution can cause in growth of granulation tissue into alvels exudate. Intra-alveoler Plugs of granulation tissue CORGANIZING PNEUMONIAE Inc. Alveolas fibrosis shrukem Firm loka 3. SPREAD OF INFECTION TO PLEURAL CAVITY • PLEURITIS: Inflammatio of Plutra) due to pratension of Inflammation from lung. undego organization, fibrous thicken of pen adhesions • PLEURAL EFFUSION -Fluid in the pleural space. • PYRO THORAX (Pus in the pleurd spre) • EMPHYSEMA collection of pes withon walls and may also result fore the persistence of PYROTHORAX. 4. BACTERIAL dissemination (BACTEREMIA) • Endocord His (Hest Valves) • pericarditis (Pericordium) • Meningitis (menges) • seppurative Artarilis (Joint) CUNICAL FEATURES D sudden onset of high fever, chills, & cough with muco perulent sputum. • FERING SUPPURATIVE PLEURITIS cause Plewiha pan. *Radiography:- Inu lake appears radio opaque i lobar jinoumoniae & focal opacities Brminsproumoniae.