University:
Camosun College
Course:
BIOL 252 | Pathophysiology for Nursing 1
Academic year:

2024
Views:

159

Pages:

3
Author:

Aysha A.

THIAZIDES cher thiazide hychlorothiazode dumen. Nat chlor Mabrlome Interstitium eally distal tubule Nat ATPA Nat Thiazide site of Action: early distal kabull 7 Nat 2 ct excretion Thiazides ✓ Ca2+ exeretion HOD Hypertension trent HT hydrochlorothiazide & chlorthalidone I Commonly wed Hypercalciuria: • & urinaly excretion of Cazd I chance of kidney Stine formation (3) aedema: Metulazone + wop dimetiocs. L in severe Cardiac failure abetre ausspidus: AB Same as wup. dimmetics. may cause mypoteme (Aldusterine Antagonists K+ Sporting drumetics Speronolactone Cantagonist MR + ALD'S • Spironolactone ceplerenone. MR: moneralvid recepta sap: Mdosterone snduyd Friten aldustesume Caginest) provents formation of Alp. ↓ *Nat excretion ases: *et retention In vedematous condition related to Aldosteroidism (Cer, hepatic corrhosis nephrotic syndrome) Competitively Sporomilatome, blocks MR Rucer: Sporonolactone used on nud. to severe heant fasture Speronulactine with thrazode thap diuretics Kt level maintained & Antihypertensive effect anhanced. Resistant hypertension to primary hyperaldosterorism.. Alchyperkalemia - magor adverse effect risk wishes impatient recently with renal disease Hause, romiting, diavilwer, faptic ulcer, gyraewmattia, o name Adverse drug interaction ACE inhibitor & SPIRONOLACTONE dampewris hyperkalemia