Dermatology Week #1

* cute urticariat urticaria ↳ erythematous - , blanching non-painful + Erythroderma of skin 195 % - . of Angio-oedema-sudden swelling · * the ↳ membrane. mucos/subdermis EX involved. is FHx blood : Ind or on ↳ skin Severe nothing ↳ holding is area Could suffer severe ↳ Patients with cancer plan a Assessment ↳ % total of body into but reside sign-blister involvement than 50yr in masor risk dermis papillary (flut rash old . , in slightly factors epidermal-dermal function rused spot) + blisters. apoptosis ... + bullue. when gently rubbed . . herpeticum Eczema ↳ HSV1 ↳ are on teratinod eczema develop Mucosul more antibiotics , Due to silimar to Nilcolskly's it cuse Appears like papelomal ↳ may 10230 . epidermis separation ↳ * Looks be could necrosis involvement ↳ 2 area . . ↳ TEN >30 % " body of total complication fulls off. skin = or HSV-2 intestion , Cluster of blister + erosion fever + cluster of blisters ↳ Superimpose ↳ Could be Signs and ↳ Spreads ↳ Comes of seen previous/pre-existing with conditions infants when low skin in Symptoms over Skin - for fever Cluster of 7-10 , days swollen + . lymph mode + Cupoptic dermatitis immunity painful Itchy (degranulation food trigger may gen . antihistamine -> Histamin ↳ causes binds unsolidation , with antihistamine , is able to to H1 receptor red - first line of defence againsturticaria + anyeodema necrosis mucocutaneous onto skin with Toxic epidermal ↳ with detachment <10 % ↳ (Toxic epidermal syndrome alteration cell drugs , 1 Stevens-Johnson's must or food/ding trigger indicating skin prick test. recent insect : MX exposure bite Iy-E response commonly food ↳ residual marking no allergic Usually lesion. prutic , within 24hr resolves * angioedema blisters common in face and neck. mucocutaneous : TBSA : area Total with body skin and surface area mucosal layer meet each other decrease this Cutuneous vasculities Purpura Cutuneous munifestation ↳ inflammation ↳ Signs non-blunching Vasculitic disorder in . Symptoms Purpura classic pulpable purpura ↳ into skin skin (small blood vessels in + bleeding = = vasculitis. > - Manuyments treatment ↳ ↳ Steroids ↳ DMARDs chronic - thats , Staphylococcal detatement mostly ↳ ↳ ↳ of also may toxigenic ↳ 3 S . (it offending disorder skin present) exotoxin release - from Staphylococcus Staphylococcus aureus aureus SJS epidermis less than children on needed. Syndrome (SSSS) blistering Superficial ↳ Skin Scaled if cases Resting Removing drug ↳ vasculities underlying in happen immunocomprimised to produce will aureus old 5year (2-3 old year at peak) 5555 adult Scaled , burned Caused : TEN : : : TBSA by 730 % severe of toxin mediated leading appearance. drug to superficial epidermal blistening. reaction epidermal detatchment blistering exfoliative A/B desmoglein[ binds to breakdown epidermis detachment. > - ↓ Large fluid filled blisters . ↳ Skin falls off ↳ Nikolosky's sign positive (sloughed in sheet (burned . Investigation - Skin and wound sumb needed when biopsy Muragement fluclocilin ceftriaxone 4 Skin , blood culture for other is causation appearance) skin with pressure Sepsis sepsis organ function dystunction shappens when infection concerned is : host by dysregulated occurs > -> inflammation response upon endothelial > - damage infection. -> congulation > - , IVF , Necrotising fusicsities pain : relief Gentle , life threatening TypeI polymicrobial infection by Type I monomicrobial , Infection spread along : : subcutaneous tissue infection suspected patient end organ dumay as supportive care. . anaerobe commonly in Streptococcus and pyogene Methicillin Resistant Staphylocus pyogene mucromicosis , fusial plane must be , does tested or in not alter surgically Finger test Poor it could be used cares freshwater infection fungal Investigation ↳ : monomicrobial Type IV * infection monomicrobial : skin . : Type I mushing shock (50-70%) as muscle layer soon as Finger . possible . L test : make 2 cm minimalistic deep resistance , incision to absence of deep bleeding fusia , . presence of necrotic tisone . Melanoma ↳ : tumor from malignant melanocytes . FHx PMH , ↳ (6mm ) spreading superficul ↳ 10 %- 80 % + Nodular melanoma larger ↳ Slow growing ↳ % Palms soles , ↳ then less Seborrhoeic over "more ↳ wort : benign torso and than like , 50 - in Sun and pigmented it grows 1cm the dermis in the early phase . , palms of time surface exposed darker skin , nail in dingnosis - hard with forehead old , into flat pigmented mucules for duration skin tumor texture vertically amelatonic growth larger thanSmm years sometimes apparatus nail beds and lesion pigmented ↳ remain may , , since slower 5-15 % larger in diagnosis Centigno Melanoma 1-3 Aerial Any diagnosis lesion > * prognosis raised modules melanoma into dermis going foor point of at Lentigo Mallyna ↳ 9-15 % rupidly growing ↳ before Superficial spread , flat light and skinned individual thread-like (fillform) early especially it patient have darker suspected changing shapes and sizes to detect are as skintone . melanoma .